Post-transplantation lymphoproliferative disease

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Lymphoproliferative disease post-renal transplantation.

Psychosocial influences on mortality after myocardial infarction. suffering, morbidity and mortality of the patients N Engl J Med 1984; 311: 552–559 diminished. 9. Covinsky KE, Kahana E, Chin MH, Palmer RM, Fortinsky RH, Landefield CS. Depressive symptoms and 3-year mortality in older hospitalized patients. Ann Intern Med 1999; 130: 563–569 References 10. Kimmel PI, Weihs KL, Peterson RA. Survi...

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Post Transplant Lymphoproliferative Disorders After Liver Transplantation

Post-transplant lymphoproliferative disorder (PTLD) is a clearly recognized and potentially life threatening complication after solid organ or bone marrow transplantation. It comprises a spectrum of diseases ranging from infectious mononucleosis and lymphoid hyperplasia to highly aggressive lymphoma. The disease has increased clinical importance in view of the constantly rising number of organ ...

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Post-transplantation lymphoproliferative disorder with gastrointestinal involvement

Post-transplantation lymphoproliferative disorders (PTLDs) are lymphoid proliferations or lymphomas that are the second most common tumors in adult transplant recipients. Most cases of PTLD are attributed to Epstein-Barr virus, which induces B-cell proliferation and occurs in the setting of severe immunosuppression after solid organ or bone marrow transplantation. The disorder is seen in 1-3% o...

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Post-transplantation lymphoproliferative disorder in pediatric patient.

Immunosuppressive therapy for transplanted patients exposes them to a high risk of developing posttransplantation lymphoproliferative disorders (PTLD). We report the case of a child undergoing heart transplantation at seven months of age who developed PTLD at nine years of age, diagnosed by resection of a pulmonary nodule.

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Posttransplant Lymphoproliferative Disease after Lung Transplantation

Posttransplant lymphoproliferative disease (PTLD) after lung transplantation occurs due to immunosuppressant therapy which limits antiviral host immunity and permits Epstein-Barr viral (EBV) replication and transformation of B cells. Mechanistically, EBV survives due to latency, escape from cytotoxic T cell responses, and downregulation of host immunity to EBV. Clinical presentation of EBV may ...

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ژورنال

عنوان ژورنال: QJM

سال: 1997

ISSN: 1460-2393

DOI: 10.1093/qjmed/90.8.497